School of Medicine
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Alexander D. Kaiser
Research Engineers, Pediatrics - Cardiology
BioAlexander D. Kaiser is a computational scientist and applied mathematician who researches modeling and simulation of heart mechanics. His doctoral work focused on the mitral valve. He currently works in the Stanford Cardiovascular Biomechanics Computation Laboratory, led by Alison Marsden, on modeling cardiac disease.
Neil M. Kalwani
Clinical Instructor, Medicine - Cardiovascular Medicine
BioNeil Kalwani, MD, MPP is a Clinical Instructor in the Division of Cardiovascular Medicine and Director of Preventive Cardiology at the VA Palo Alto Health Care System. He attended college at Yale University and completed graduate degrees in medicine and public policy at Harvard University. He trained in internal medicine at Brigham and Women’s Hospital before arriving at Stanford in 2018 for fellowship in cardiovascular medicine, during which he served as Chief Fellow. He then completed a postdoctoral fellowship through the Stanford-AHRQ Health Services Research Training Program in the Department of Health Policy. His clinical focus is in general and preventive cardiology and echocardiography. He practices at the VA Palo Alto Health Care System and at Stanford Health Care.
Dr. Kalwani's research focuses on the evaluation of policies and care delivery innovations designed to improve the value of care for patients with cardiovascular disease.
Clinical Assistant Professor, Medicine - Cardiovascular Medicine
BioDr. Kang is an interventional cardiologist who specializes in the treatment of structural heart disease. He has expertise in complex coronary interventions, transcatheter aortic and mitral valve replacements, transcatheter mitral valve repair, left atrial appendage occlusion, PFO/septal defect closure, alcohol septal ablation, and paravalvular leak closure.
A Bay Area native, he graduated from Stanford University and obtained his medical degree at Yale University. He came back to Stanford to train in internal medicine, cardiology, and interventional cardiology before completing an advanced structural interventions fellowship at Ford Hospital.
Associate Professor of Medicine (Pulmonary and Critical Care Medicine)
Current Research and Scholarly InterestsOur research program has several active projects:
1.) Pulmonary Vascular Disease Simvastatin reversed experimental pulmonary hypertension, and is safe for treatment of patients. Blinded clinical trials of efficacy are in progress.
2.) Lung inflammation and regeneration (stem cells)
3.) Lung surfactant rheology and oxidative stress
4.) Gene regulation by RNA binding proteins, NF45 and NF90 through transcriptional and posttranscriptional mechanisms
Michael S. Kapiloff, MD, PhD
Reinhard Family Professor, Professor (Research) of Ophthalmology and, by courtesy, of Medicine (Cardiovascular Medicine)
Current Research and Scholarly InterestsDr. Michael S. Kapiloff is a faculty member in the Departments of Ophthalmology and Medicine (Cardiovascular Medicine) and a member of the Stanford Cardiovascular Institute. Although Dr. Kapiloff was at one time a Board-Certified General Pediatrician, he is currently involved in full-time basic science and translational research. His laboratory studies the basic molecular mechanisms underlying the response of the retinal ganglion cell and cardiac myocyte to disease. The longstanding interest of his laboratory is the role in intracellular signal transduction of multimolecular complexes organized by scaffold proteins. Recently, his lab has also been involved in the translation of these concepts into new therapies, including the development of new AAV gene therapy biologics for the prevention and treatment of heart failure and for neuroprotection in the eye.
URL to NCBI listing of all published works:
For more information see Dr. Kapiloff's lab website: http://med.stanford.edu/kapilofflab.html
Associate Professor (Research) of Cardiothoracic Surgery
Current Research and Scholarly InterestsThe Karakikes Lab aims to uncover fundamental new insights into the molecular mechanisms and functional consequences of pathogenic mutations associated with familial cardiovascular diseases.