School of Medicine
Showing 21-30 of 32 Results
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Palca Shibale
Postdoctoral Scholar, Plastic and Reconstructive Surgery
BioShibale, Palca is a post-doctoral fellow in the Hagey Laboratory under mentorship of Dr. Derrick Wan and Michael Longaker. She earned her BS in Molecular and Cellular Biology at the University of Washington (UW), her MS in Medical Physiology and Biophysics at Case Western University and her MD from UW. She has previously conducted translational research on drug efficacy and clinical research in trauma and vascular surgery. Her current works focus on understanding the mechanisms of tissue regeneration and fibrosis with nano materials and as well, the roles of fibroblast subpopulations in the foreign body response model
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Ken Suzuki
Postdoctoral Scholar, General Surgery
BioKen Suzuki is a postdoctoral researcher at Stanford University's Department of Surgery. He received his PhD in Economics from the University of California at Santa Cruz in June 2024. Before UC Santa Cruz, he earned his MA in Economics from Hitotsubashi University in 2017 and his BA in Economics from Yokohama National University in 2015. His research interests include health economics, applied microeconomics, econometrics, and statistical causal inference. His research utilizes large administrative datasets from the Veterans Health Administration (VA) to examine how health policies affect patient healthcare utilization and health outcomes.
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Humza Thobani
Postdoctoral Scholar, Pediatric Surgery
BioHumza is a Postdoctoral Research Fellow in the Division of Pediatric Surgery at Stanford University. He earned his medical degree from the Aga Khan University in Karachi, Pakistan in 2023. Prior to joining Stanford, he had completed a dedicated research fellowship in pediatric surgery, also at the Aga Khan University, where he was named Best Research Fellow in 2024.
Humza's research interests revolve around congenital surgical anomalies, pediatric solid tumors, and pediatric inflammatory bowel diseases, with a focus on leveraging big data and machine learning methods to study rare pediatric conditions. -
Antonio Tomasso
Postdoctoral Scholar, Plastic and Reconstructive Surgery
BioAntonio Tomasso is an NWO Rubicon Postdoctoral Scholar. As part of his MSc in Medical Molecular and Cellular Biotechnology at Vita-Salute San Raffaele University, he explored the immunomodulatory and neurotrophic roles of neural stem cells (NSCs) following spinal cord injury. As a Research Assistant at Karolinska Institute, he delved into the signaling pathways required for NSC activation and migration after spinal cord injury, and the limited regenerative abilities of mouse and human heart.
During his PhD, he investigated the molecular mechanisms of tissue regeneration in planarians, axolotls and spiny mice. He conducted research as a Visiting Fellow at the University of Kentucky and the Hubrecht Institute.
His research demonstrated that MAPK/ERK signaling acts as a molecular switch between regeneration and fibrosis in adult mammals and can be activated to stimulate a regenerative response, including new hair follicle formation, in scarring wounds.
He contributed to a pioneering study showing that spiny mice can recover heart function after infarct through enhanced angiogenesis, ECM remodeling and epicardium regeneration. He also played a key role in spatial transcriptomic studies that defined regenerative and fibrotic gene signatures in spiny mice, laboratory mice and gerbils.
He earned a PhD cum laude in Molecular and Cellular Life Sciences at the Max Planck Institute for Molecular Biomedicine.
He has been awarded an NWO Dutch Research Council Rubicon Postdoctoral grant to conduct research on the molecular drivers of fibroblast activation in wound healing and organ fibrosis.
His ultimate research aim is to crack the code of tissue regeneration and rejuvenation, reversing organ scarring and preventing fibrosis in injuries and pathological conditions, through the identification of therapeutic targets for enhanced tissue repair and functional recovery.
Driven by his innate curiosity and passion for science, he loves tackling new challenges, thinking outside the box, and building interdisciplinary collaborations to push forward the boundaries of knowledge.
His career goal is to serve as a group leader, fostering a collaborative environment where everyone can thrive, achieve their goals and leave a lasting impact through community-building and scientific discoveries for the benefit of humankind. -
Banita Verma
Postdoctoral Scholar, General and Vascular Surgery
BioI am Banita Verma, a postdoctoral researcher at Stanford University, currently working with Dr. Fredrick M. Dirbas at the Department of Surgery. Our research focus is to understand the type of cell death and the nature of immune responses triggered by FLASH versus conventional radiotherapy in various murine breast cancer models. Additionally, we aim to explore the role of DAMPs released by dying cells in generating immune responses after both FLASH and conventional radiotherapy. Furthermore, we are interested in studying the mechanism behind the low toxicity of the FLASH compared to conventional radiotherapy to the adjacent non-cancerous tissue. To accomplish this goal, our laboratory is actively collaborating with Dr. Bill Loo, who holds great expertise in the field of radiation oncology.
Before joining Stanford University, I served as a postdoctoral researcher at Karolinska Institutet, Sweden from 2021-2023. My research aimed to study the activation mechanism of Cholineacetyltransferase (ChAT), a pivotal enzyme in acetylcholine synthesis. This enzyme is known to be hypoactive in neurodegenerative conditions such as Alzheimer's and Parkinson's disease. Our group successfully synthesized novel compounds capable of enhancing ChAT activity.
I completed my doctoral research in cancer biology at the Department of Experimental Medicine and Biotechnology, PGIMER Chandigarh, India, in 2021. My work was the evaluation of role of TNF-α mediated Necroptosis in breast cancer cells. My primary research interests are cancer biology and cell death pathways.
