Dr. Gillian Heinecke joined Stanford Medicine in August 2017 as a Clinical Assistant Professor of Dermatology. She earned a Bachelors of Science in Biology from Brown University in 2007 and a medical degree with distinction in research from Icahn School of Medicine at Mount Sinai in 2013, where she was also elected to the Alpha Omega Alpha society. She completed a one year research fellowship in biologic treatment for psoriasis with Dr. Mark Lebwohl while at Mount Sinai. In 2017, Dr. Heinecke completed residency training in dermatology at Stanford where she served as Chief Resident. She is board certified in dermatology by the American Board of Dermatology.

Her professional interests include psoriasis, complex medical dermatology, skin cancer, infectious diseases of the skin and medical education.

Clinical Focus

  • Psoriasis
  • Complex Medical Dermatology
  • Skin Cancer
  • Infectious Diseases of the Skin
  • Dermatology

Academic Appointments

Professional Education

  • Medical Education: Icahn School of Medicine at Mount Sinai (2013) NY
  • Board Certification: Dermatology, American Board of Dermatology (2017)
  • Residency: Stanford University Dermatology Residency (2017) CA
  • Internship: Kaiser Permanente Oakland Internal Medicine Residency (2014) CA

All Publications

  • Intraepidermal Type VII Collagen by Immunofluorescence Mapping: A Specific Finding for Bullous Dermolysis of the Newborn. Pediatric dermatology Heinecke, G., Marinkovich, M. P., Rieger, K. E. 2017; 34 (3): 308-314


    Bullous dermolysis of the newborn (BDN) is a subtype of dystrophic epidermolysis bullosa (DEB) characterized by skin fragility and blister formation at birth that typically resolves within the first year of life. Abnormal intraepidermal retention of type VII collagen (C7) has been reported as a characteristic feature of BDN, but few studies have investigated the specificity of this finding.We retrospectively reviewed pathology reports of patients diagnosed with DEB using immunofluorescence mapping from January 2001 to January 2015. For cases describing intraepidermal accumulation of C7, we collected information on patient characteristics, including genetic testing results, clinical outcome, and concurrent electron microscopy findings, where available.Of the 143 cases of DEB with immunofluorescence mapping, eight patients had intracytoplasmic epidermal retention of C7. Of these eight patients, two were lost to follow-up, four had complete resolution of bullae, and two had marked improvement with rare residual bullae. Concurrent electron microscopic findings available for three patients were consistent with BDN.Our review of immunofluorescence mapping findings in patients with DEB found that 5.6% had abnormal intracytoplasmic epidermal retention of C7, a finding previously reported in BDN. All such patients with clinical outcomes available had resolution or marked improvement of bullae, consistent with clinical outcomes expected in BDN.

    View details for DOI 10.1111/pde.13132

    View details for PubMedID 28523885