Honors & Awards

  • Rising Environmental Leaders Program, Stanford Woods Institute for the Environment (2015)
  • Center for a Livable Future Lerner Doctoral Fellowship, Johns Hopkins University Bloomberg School of Public Health (2009-2013)

Boards, Advisory Committees, Professional Organizations

  • Member, International Society for Environmental Epidemiology (2014 - Present)
  • Member, American Public Health Association (2010 - Present)
  • Certified Industrial Hygienist, American Board of Industrial Hygiene (2005 - Present)

Professional Education

  • Master of Science, Johns Hopkins University (2000)
  • Doctor of Philosophy, Johns Hopkins University (2013)
  • Bachelor of Arts, University of California Berkeley (1995)

Stanford Advisors

Current Research and Scholarly Interests

Dr. Hartle's research investigates environmental exposures and their effects on human health, with a special focus on environmental exposures from the food system. Her research interests are in exposure science, risk assessment, and environmental epidemiology and how they can be used to inform environmental health and food system policy. Jennifer's doctoral research at Johns Hopkins focused on identifying inequities in dietary environmental exposures. For her dissertation, she examined bisphenol-A (BPA) exposures in the food system to determine vulnerable populations and to develop exposure prevention strategies. Dr. Hartle's current research projects explore environmental endocrine disrupting chemicals and obesity.

All Publications

  • Probabilistic modeling of school meals for potential bisphenol A (BPA) exposure. Journal of exposure science and environmental epidemiology Hartle, J. C., Fox, M. A., Lawrence, R. S. 2016; 26 (3): 315-323


    Many endocrine-disrupting chemicals (EDCs), including bisphenol A (BPA), are approved for use in food packaging, with unbound BPA migrating into the foods it contacts. Children, with their developing organ systems, are especially susceptible to hormone disruption, prompting this research to model the potential dose of BPA from school-provided meals. Probabilistic exposure models for school meals were informed by mixed methods. Exposure scenarios were based on United States school nutrition guidelines and included meals with varying levels of exposure potential from canned and packaged food. BPA exposure potentials were modeled with a range of 0.00049 μg/kg-BW/day for a middle school student with a low exposure breakfast and plate waste to 1.19 μg/kg-BW/day for an elementary school student eating lunch with high exposure potential. The modeled BPA doses from school meals are below the current US EPA Oral Reference Dose (RfD) of 50 μg/kg-BW/day. Recent research shows BPA animal toxicity thresholds at 2 μg/kg-BW/day. The single meal doses modeled in this research are at the same order of magnitude as the low-dose toxicity thresholds, illustrating the potential for school meals to expose children to chronic toxic levels of BPA.

    View details for DOI 10.1038/jes.2015.58

    View details for PubMedID 26395857

  • Weight loss on low-fat vs. low-carbohydrate diets by insulin resistance status among overweight adults and adults with obesity: A randomized pilot trial OBESITY Gardner, C. D., Offringa, L. C., Hartle, J. C., Kapphahn, K., Cherin, R. 2016; 24 (1): 79-86

    View details for DOI 10.1002/oby.21331

    View details for Web of Science ID 000367189800014

  • The consumption of canned food and beverages and urinary Bisphenol A concentrations in NHANES 2003–2008 Environmental Research Hartle, J. C., Navas-Acien, A., Lawrence, R. S. 2016; 150: 375-382
  • Early Life Metabolism of Bisphenol A: A Systematic Review of the Literature. Current Environmental Health Reports 2014 Nachman, R. M., Hartle, J. C., Lees, P. S., Groopman, J. D. 2014; 1 (1)
  • Bisphenol A: A ubiquitous food system contaminant Introduction to the U.S. Food System: Public Health, Environment, and Equity Hartle, J. C. Jossey-Bass, An Imprint of John Wiley & Sons, Inc. . 2014
  • A comparative study of allowable pesticide residue levels on produce in the United States GLOBALIZATION AND HEALTH Neff, R. A., Hartle, J. C., Laestadius, L. I., Dolan, K., Rosenthal, A. C., Nachman, K. E. 2012; 8


    The U.S. imports a substantial and increasing portion of its fruits and vegetables. The U.S. Food and Drug Administration currently inspects less than one percent of import shipments. While countries exporting to the U.S. are expected to comply with U.S. tolerances, including allowable pesticide residue levels, there is a low rate of import inspections and few other incentives for compliance.This analysis estimates the quantity of excess pesticide residue that could enter the U.S. if exporters followed originating country requirements but not U.S. pesticide tolerances, for the top 20 imported produce items based on quantities imported and U.S. consumption levels. Pesticide health effects data are also shown.The model estimates that for the identified items, 120 439 kg of pesticides in excess of U.S. tolerances could potentially be imported to the U.S., in cases where U.S. regulations are more protective than those of originating countries. This figure is in addition to residues allowed on domestic produce. In the modeling, the top produce item, market, and pesticide of concern were oranges, Chile, and Zeta-Cypermethrin. Pesticides in this review are associated with health effects on 13 body systems, and some are associated with carcinogenic effects.There is a critical information gap regarding pesticide residues on produce imported to the U.S. Without a more thorough sampling program, it is not possible accurately to characterize risks introduced by produce importation. The scenario presented herein relies on assumptions, and should be considered illustrative. The analysis highlights the need for additional investigation and resources for monitoring, enforcement, and other interventions, to improve import food safety and reduce pesticide exposures in originating countries.

    View details for DOI 10.1186/1744-8603-8-2

    View details for Web of Science ID 000302059900001

    View details for PubMedID 22293037