Dr. DiBiase is an obstetrician and gynecologist with the Stanford Health Care Gynecology Clinic in Palo Alto. She is also a clinical assistant professor in the Department of Obstetrics and Gynecology.
Dr. DiBiase specializes in providing comprehensive, holistic obstetric and gynecologic care for patients from adolescence through menopause. She aims to provide patient-centric, equitable, and excellent care. Her area of clinical interests includes minimally invasive gynecologic surgery, family planning care, and patient advocacy. Dr. Dibiase also brings a global perspective to her medical practice. Her training and experience include delivering a broad range of medical care to diverse populations in Vietnam, as well as providing OB-GYN services to women in Uganda.
Dr. DiBiase’s clinical research interests include improving care of patients with substance use disorders in pregnancy and while breastfeeding. She is also currently engaged in developing training curricula for medical students, providers, and hospital staff to increase awareness of and combat racism in reproductive health care settings. Her aim is to help clinicians foster inclusive care environments that provide antiracist, equitable care to all patient demographics.
Dr. DiBiase has published her work in peer-reviewed journals and presented at multiple national meetings. She also recently coauthored a chapter in the third edition of Pocket Obstetrics and Gynecology, a widely used reference for OB-GYN trainees and practitioners.
- Obstetrics and Gynecology
- Gynecologic Surgery
- Prenatal and Obstetric Care
- Contraception and Family Planning
- Abnormal Uterine Bleeding
- Substance Use Disorders in Pregnancy
- Preventative Care
- LGBTQ+ Care
Clinical Assistant Professor, Obstetrics & Gynecology - Maternal Fetal Medicine
Honors & Awards
Donald R. Coustan, M.D. Courage to Lead Award, Brown University/Women & Infants Hospital (2023)
Academic Chief Resident, Brown University/Women & Infants Hospital (2022-2023)
AUGS Award for Excellence in Female Pelvic Medicine and Reconstructive Surgery, Brown University/Women & Infants Hospital (2022)
Positive Champion of the Learning Environment, Brown University/Women & Infants Hospital (2020)
Charles T. Schechtman, M.D. ’29 Prize for excellence in the clinical care of patients, University of Vermont Larner College of Medicine (2019)
Carabee Award for academic excellence in obstetrics, gynecology, and reproductive sciences, University of Vermont Larner College of Medicine (2019)
Helen and Phyllis Wasserman Phorplus Prize for Excellence in the Basic Sciences, University of Vermont Larner College of Medicine
Boards, Advisory Committees, Professional Organizations
Junior Fellow in Practice, American College of Obstetrics and Gynecology (2019 - Present)
Residency: Women and Infants Hospital of Rhode Island Obstetrics and Gynecology Residency RI
Medical Education: University of Vermont College of Medicine (2019) VT
BA, Middlebury College (2013)
Additional Clinical Info
Cannabis Use in Pregnancy: Effects of a Hospital Policy on Toxicology Screening Practices
LIPPINCOTT WILLIAMS & WILKINS. 2023: 5S
View details for Web of Science ID 001043263400017
- Cervical ectopic and cesarean scar pregnancies: navigating the differential of an abnormal lower uterine pregnancy Topics in Obstetrics and Gynecology 2023
ARNT2 Tunes Activity-Dependent Gene Expression through NCoR2-Mediated Repression and NPAS4-Mediated Activation.
2019; 102 (2): 390-406.e9
Neuronal activity-dependent transcription is tuned to ensure precise gene induction during periods of heightened synaptic activity, allowing for appropriate responses of activated neurons within neural circuits. The consequences of aberrant induction of activity-dependent genes on neuronal physiology are not yet clear. Here, we demonstrate that, in the absence of synaptic excitation, the basic-helix-loop-helix (bHLH)-PAS family transcription factor ARNT2 recruits the NCoR2 co-repressor complex to suppress neuronal activity-dependent regulatory elements and maintain low basal levels of inducible genes. This restricts inhibition of excitatory neurons, maintaining them in a state that is receptive to future sensory stimuli. By contrast, in response to heightened neuronal activity, ARNT2 recruits the neuronal-specific bHLH-PAS factor NPAS4 to activity-dependent regulatory elements to induce transcription and thereby increase somatic inhibitory input. Thus, the interplay of bHLH-PAS complexes at activity-dependent regulatory elements maintains temporal control of activity-dependent gene expression and scales somatic inhibition with circuit activity.
View details for DOI 10.1016/j.neuron.2019.02.007
View details for PubMedID 30846309
View details for PubMedCentralID PMC6504996