Clinical Focus

  • Anesthesiology

Academic Appointments

Professional Education

  • Medical Education: Stanford University School of Medicine (1994) CA
  • Board Certification: American Board of Anesthesiology, Anesthesiology (1999)
  • Residency: UCSF Anesthesiology Residency (1998) CA
  • Internship: Alameda County Highland Hospital Internal Medicine Residency (1995) CA

Contact

  • Academic
    hchow@stanford.edu
    University - Academic staff Department: Anesthesia Position: Clinical Associate Professor
    • 300 Pasteur Dr Rm H3580
    • Department Of Anesthesia
    • Stanford,  California  94305-2200 
  • Clinical (Primary)  Anesthesia Department 300 Pasteur Dr Rm H3580 MC 5640 Stanford, CA 94305 (650) 725-8544 (fax)

Additional Clinical Info

Additional Info

  • Mail Code: 5640

All Publications

  • Anesthetic-Induced Intraoperative Dream Associated With Remission of a Psychiatric Disorder: A Case Report. A&A practice Chow, H. S., Hack, L. M., Kawai, M., Heifets, B. D. 2022; 16 (8): e01613

    Abstract

    Trauma is associated with debilitating acute and posttraumatic stress disorders, which have limited treatment options. We report on a patient undergoing surgical hand repair after a recent knife attack who experienced vivid dreaming and subsequent remission of acute stress disorder. After local anesthesia with propofol sedation she recalled a dream wherein she relived the attack, sought medical care, completed surgery, and returned home with a healed hand. While intraoperative dreaming is common, this case details potential associations between anesthetic state, dreaming, intraoperative electroencephalography, and remission of a psychiatric disorder. Our experience suggests a novel intervention for stress disorders.

    View details for DOI 10.1213/XAA.0000000000001613

    View details for PubMedID 35952341

  • TROLOX ATTENUATES CORTICAL NEURONAL INJURY-INDUCED BY IRON, ULTRAVIOLET-LIGHT, GLUCOSE DEPRIVATION, OR AMPA BRAIN RESEARCH Chow, H. S., Lynch, J. J., Rose, K., Choi, D. W. 1994; 639 (1): 102-108

    Abstract

    The vitamin E analog, trolox, protected cultured cortical neurons against damage induced by exposure to either iron ions or ultraviolet (UV) light, consistent with an ability to inhibit free radical-mediated cytotoxicity. Trolox also reduced neuronal death induced by 24 h exposure to alpha-amino-3-hydroxy-5-methyl-4-isoxazole propionic acid (AMPA), but not that induced by N-methyl-D-aspartate (NMDA). When combined with the NMDA receptor antagonist dextrorphan, trolox also reduced the neuronal injury induced by glucose deprivation.

    View details for Web of Science ID A1994MZ35700013

    View details for PubMedID 7514085