Dr. Logan Schneider specializes in the treatment of sleep disorders, which include things like sleep apnea, narcolepsy, insomnia, restless legs syndrome, sleepwalking, and REM-sleep behavior disorder. He has practiced Sleep Neurology for more than 5 years. Dr. Schneider has a special interest in REM-sleep behavior disorder and other parasomnias (such as sleepwalking).

Clinical Focus

  • Sleep Medicine

Administrative Appointments

  • Chair, Alliance Awards Workgroup, American Academy of Neurology (2017 - Present)
  • Project Leader, New Program Offerings workgroup, American Academy of Neurology - Graduate Education Subcommittee (2017 - 2018)
  • Chair, Sleep Medicine Section, American Academy of Neurology (2016 - 2020)
  • Chair-elect, Chair, Past Chair (3-year term), Consortium of Neurology Residents and Fellows, American Academy of Neurology (2013 - 2016)
  • Chief Resident, Johns Hopkins, Department of Neurology (2013 - 2014)
  • Department of Neurology Representative, Housestaff Patients Safety and Quality Council, Johns Hopkins Medical Institute (2013 - 2014)
  • Webmaster, Neurology Residency website, Johns Hopkins, Department of Neurology (2013 - 2014)
  • Resident representative, Residency Selection Committee, Johns Hopkins, Department of Neurology (2012 - 2014)

Honors & Awards

  • SRS Trainee Merit Award, Sleep Research Society (2017)
  • AASM Young Investigators Research Forum, American Academy of Sleep Medicine (2016)
  • AAN Fellow Scholarship to the Annual Meeting, American Academy of Neurology (2015)
  • Sleep Research Network Travel Award, Sleep Research Network (2015)
  • AAN Resident Scholarship to the Annual Meeting, American Academy of Neurology (2014)
  • Housestaff Teaching Award, Johns Hopkins Medical Institute (2014)

Boards, Advisory Committees, Professional Organizations

  • AAN Annual Meeting Sleep Topic workgroup member, American Academy of Neurology (2017 - Present)
  • AASM Trainee Symposia Series Subcommittee member, American Academy of Sleep Medicine (2017 - Present)
  • Editorial Board member, Practical Neurology (2017 - Present)
  • Educational Products Subcommittee member, American Academy of Sleep Medicine (2016 - Present)
  • Graduate Education Subcommittee member, American Academy of Neurology (2015 - Present)
  • Awards Workgroup member, American Academy of Neurology (2015 - 2017)

Professional Education

  • Doctor of Medicine, University of Florida (2010)
  • Board Certification: Sleep Medicine, American Board of Psychiatry and Neurology (2015)
  • Board Certification: Neurology, American Board of Psychiatry and Neurology (2014)
  • Fellowship:Stanford Hospitals and Clinics (2015) CA
  • Residency:Johns Hopkins Hospital and Bayview Medical Center (2014) MD
  • Internship:Shands at the University of Florida (2011) FL
  • Medical Education:University of Florida, College of Medicine (2010) FL

Stanford Advisors

Community and International Work

  • Manual of the Neurologic Examination for Neurologists in Training


    Neurology clinical training

    Partnering Organization(s)

    European Academy of Neurology and German Neurological Society

    Populations Served

    Neurologist and physicians-in-training throughout the world



    Ongoing Project


    Opportunities for Student Involvement


  • Synopsys Science & Technology Championship, Santa Clara, CA


    middle/high school science fair competition

    Populations Served

    Students in Silicon Valley


    Bay Area

    Ongoing Project


    Opportunities for Student Involvement


  • Cardinal Free Clinics


    Free general and neurologic medical care for the local community

    Partnering Organization(s)

    Stanford University, School of Medicine

    Populations Served

    Bay Area indigent population


    Bay Area

    Ongoing Project


    Opportunities for Student Involvement


Current Research and Scholarly Interests

From a research perspective, my long-term career plan is to refine the understanding of normal and dysfunctional sleep, much like the Epilepsy Phenome/Genome Project (EPGP) and Epi4K are doing for the enigmatic epilepsies. Insufficient sleep has been deemed a public health problem with poorly understood behavioral and physiologic sleep disorders lying at the core of the issue. I am currently using well-defined distinct and objective phenotypes (e.g. periodic limb movements, hypocretin-deficient narcolepsy) to acquire the analytic skills necessary to expand my knowledge of both signal processing and genetics, with the former enhancing my ability to identify and/or refine sleep phenotypes, and the latter facilitating the pathophysiological understanding of these phenotypes. As a consequence of a better link between symptoms/phenotypes, physiology, and genetic risks, more personally targeted and effective therapeutics can be developed to address the enriched spectrum of sleep disorders.

Graduate and Fellowship Programs

All Publications

  • Automatic, ECG-based detection of autonomic arousals and their association with cortical arousals, leg movements, and respiratory events in sleep. Sleep Olsen, M., Schneider, L. D., Cheung, J., Peppard, P. E., Jennum, P. J., Mignot, E., Sorensen, H. B. 2018


    The current definition of sleep arousals neglects to address the diversity of arousals and their systemic cohesion. Autonomic arousals (AA) are autonomic activations often associated with cortical arousals (CA), but they may also occur in isolation in relation to a respiratory event, a leg movement event or spontaneously, without any other physiological associations. AA should be acknowledged as essential events to understand and explore the systemic implications of arousals.We developed an automatic AA detection algorithm based on intelligent feature selection and advanced machine learning using the electrocardiogram. The model was trained and tested with respect to CA systematically scored in 258 (181 training size/77 test size) polysomnographic recordings from the Wisconsin Sleep Cohort.A precision value of 0.72 and a sensitivity of 0.63 were achieved when evaluated with respect to CA. Further analysis indicated that 81% of the non-CA-associated AAs were associated with leg movement (38%) or respiratory (43%) events.The presented algorithm shows good performance when considering that more than 80% of the false positives (FP) found by the detection algorithm appeared in relation to either leg movement or respiratory events. This indicates that most FP constitute autonomic activations that are indistinguishable from those with cortical cohesion. The proposed algorithm provides an automatic system trained in a clinical environment, which can be utilized to analyse the systemic and clinical impacts of arousals.

    View details for DOI 10.1093/sleep/zsy006

    View details for PubMedID 29329416

  • Genetic Basis of Chronotype in Humans: Insights From Three Landmark GWAS SLEEP Kalmbach, D. A., Schneider, L. D., Cheung, J., Bertrand, S. J., Kariharan, T., Pack, A. I., Gehrman, P. R. 2017; 40 (2)


    Chronotype, or diurnal preference, refers to behavioral manifestations of the endogenous circadian system that governs preferred timing of sleep and wake. As variations in circadian timing and system perturbations are linked to disease development, the fundamental biology of chronotype has received attention for its role in the regulation and dysregulation of sleep and related illnesses. Family studies indicate that chronotype is a heritable trait, thus directing attention toward its genetic basis. Although discoveries from molecular studies of candidate genes have shed light onto its genetic architecture, the contribution of genetic variation to chronotype has remained unclear with few related variants identified. In the advent of large-scale genome-wide association studies (GWAS), scientists now have the ability to discover novel common genetic variants associated with complex phenotypes. Three recent large-scale GWASs of chronotype were conducted on subjects of European ancestry from the 23andMe cohort and the UK Biobank. This review discusses the findings of these landmark GWASs in the context of prior research.We systematically reviewed and compared methodological and analytical approaches and results across the three GWASs of chronotype.A good deal of consistency was observed across studies with 9 genes identified in 2 of the 3 GWASs. Several genes previously unknown to influence chronotype were identified.GWAS is an important tool in identifying common variants associated with the complex chronotype phenotype, the findings of which can supplement and guide molecular science. Future directions in model systems and discovery of rare variants are discussed.

    View details for DOI 10.1093/sleep/zsw048

    View details for Web of Science ID 000394129900011

    View details for PubMedID 28364486

  • Breathing Disturbances Without Hypoxia Are Associated With Objective Sleepiness in Sleep Apnea. Sleep Koch, H., Schneider, L. D., Finn, L. A., Leary, E. B., Peppard, P. E., Hagen, E., Sorensen, H. B., Jennum, P., Mignot, E. 2017; 40 (11)


    To determine whether defining two subtypes of sleep-disordered breathing (SDB) events-with or without hypoxia-results in measures that are more strongly associated with hypertension and sleepiness.A total of 1022 participants with 2112 nocturnal polysomnograms from the Wisconsin Sleep Cohort were analyzed with our automated algorithm, developed to detect breathing disturbances and desaturations. Breathing events were time-locked to desaturations, resulting in two indices-desaturating (hypoxia-breathing disturbance index [H-BDI]) and nondesaturating (nonhypoxia-breathing disturbance index [NH-BDI]) events-regardless of arousals. Measures of subjective (Epworth Sleepiness Scale) and objective (2981 multiple sleep latency tests from a subset of 865 participants) sleepiness were analyzed, in addition to clinically relevant clinicodemographic variables. Hypertension was defined as BP ≥ 140/90 or antihypertensive use.H-BDI, but not NH-BDI, correlated strongly with SDB severity indices that included hypoxia (r ≥ 0.89, p ≤ .001 with 3% oxygen-desaturation index [ODI] and apnea hypopnea index with 4% desaturations). A doubling of desaturation-associated events was associated with hypertension prevalence, which was significant for ODI but not H-BDI (3% ODI OR = 1.06, 95% CI = 1.00-1.12, p < .05; H-BDI OR 1.04, 95% CI = 0.98-1.10) and daytime sleepiness (β = 0.20 Epworth Sleepiness Scale [ESS] score, p < .0001; β = -0.20 minutes in MSL on multiple sleep latency test [MSLT], p < .01). Independently, nondesaturating event doubling was associated with more objective sleepiness (β = -0.52 minutes in MSL on MSLT, p < .001), but had less association with subjective sleepiness (β = 0.12 ESS score, p = .10). In longitudinal analyses, baseline nondesaturating events were associated with worsening of H-BDI over a 4-year follow-up, suggesting evolution in severity.In SDB, nondesaturating events are independently associated with objective daytime sleepiness, beyond the effect of desaturating events.

    View details for DOI 10.1093/sleep/zsx152

    View details for PubMedID 29029253

  • Diagnosis and Management of Narcolepsy. Seminars in neurology Schneider, L., Mignot, E. 2017; 37 (4): 446–60

    View details for DOI 10.1055/s-0037-1605554

    View details for PubMedID 28837992

  • Education Research: Neurology resident education: Trending skills, confidence, and professional preparation. Neurology Jordan, J. T., Mayans, D., Schneider, L., Adams, N., Khawaja, A. M., Engstrom, J. 2016; 86 (11): e112-7


    To survey US-trained graduating neurology residents who are American Academy of Neurology members, in an effort to trend perceived quality and completeness of graduate neurology education.An electronic survey was sent to all American Academy of Neurology members graduating from US neurology residency programs in the Spring of 2014.Of 805 eligible respondents, 24% completed the survey. Ninety-three percent of adult neurology residents and 56% of child neurology residents reported plans to pursue fellowship training after residency. Respondents reported a desire for additional training in neurocritical care, neuro-oncology, neuromuscular diseases, botulinum toxin injection, and nerve blocks. There remains a clear deficit in business training of neurology residents, although there was notable improvement in knowledge of coding and office management compared to previous surveys.Although there are still areas of perceived weakness in neurology training, graduating neurology residents feel generally well prepared for their chosen careers. However, most still pursue fellowship training for reasons that are little understood. In addition to certain subspecialties and procedures, practice management remains deficient in neurology training and is a point of future insecurity for most residents. Future curriculum changes should consider resident-reported gaps in knowledge, with careful consideration of improving business training.

    View details for DOI 10.1212/WNL.0000000000002463

    View details for PubMedID 26976522

  • Voice of young neurologists around the world. Neurology Rakusa, M., Struhal, W., Gak, S., Tanprawate, S., Balicza, P., Khawaja, A. M., Schneider, L. D. 2016; 86 (4): e40-1

    View details for DOI 10.1212/WNL.0000000000002317

    View details for PubMedID 26810430

  • Clinical Reasoning: A 44-year-old woman with rapidly progressive weakness and ophthalmoplegia. Neurology Schreck, K. C., Schneider, L., Geocadin, R. G. 2015; 85 (3): e22-7

    View details for DOI 10.1212/WNL.0000000000001761

    View details for PubMedID 26195241

  • Clinical Reasoning: A 68-year-old man with a first presentation of status epilepticus NEUROLOGY Schneider, L., Probasco, J. C., Newsome, S. D. 2014; 82 (14): E116-E121


    A 64-year-old man with transfusion-dependent myelodysplastic syndrome (MDS), hypertension, chronic obstructive pulmonary disease, hypothyroidism, blindness from treated syphilitic chorioretinitis, and no prior seizure history presented in generalized status epilepticus. His daily home medication regimen included prednisone 20 mg (chronic therapy for MDS), diltiazem 120 mg, digoxin 250 μg, tiotropium 80 μg, and levothyroxine 112 μg. On admission he was febrile to 39.9 °C and in atrial fibrillation with rapid ventricular rate. Initial hematologic profile showed 11,910 leukocytes/mm(3) (12% immature forms, 46% neutrophils, 32% lymphocytes), hematocrit of 30.8%, and platelet count of 215,000/mm(3), with an otherwise normal serum chemistry.

    View details for DOI 10.1212/WNL.0000000000000289

    View details for Web of Science ID 000336497000002

    View details for PubMedID 24711538

  • International Issues: Obtaining an adult neurology residency position in the United States An overview NEUROLOGY Jordan, J. T., Sellner, J., Struhal, W., Schneider, L., Mayans, D. 2014; 82 (14): E112-E115


    Around the world, there are marked differences in neurology training, including training duration and degree of specialization. In the United States, adult neurology residency is composed of 1 year of internal medicine training (preliminary year) and 3 years of neurology-specific training. Child neurology, which is not the focus of this article, is 2 years of pediatrics and 3 years of neurology training. The route to adult neurology residency training in the United States is standardized and is similar to most other US specialties. Whereas US medical graduates often receive stepwise guidance from their medical school regarding application for residency training, international graduates often enter this complex process with little or no such assistance. Despite this discrepancy, about 10%-15% of residency positions in the United States are filled by international medical graduates.(1,2) In adult neurology specifically, 35% of matched positions were filled by international graduates in 2013, 75% of whom were not US citizens.(1) In an effort to provide a preliminary understanding of the application process and related terminology (table 1) and thereby encourage international residency applicants, we describe the steps necessary to apply for neurology residency in the United States.

    View details for DOI 10.1212/WNL.0000000000000279

    View details for Web of Science ID 000336497000001

    View details for PubMedID 24711537

  • What We Think before a Voluntary Movement JOURNAL OF COGNITIVE NEUROSCIENCE Schneider, L., Houdayer, E., Bai, O., Hallett, M. 2013; 25 (6): 822-829


    A central feature of voluntary movement is the sense of volition, but when this sense arises in the course of movement formulation and execution is not clear. Many studies have explored how the brain might be actively preparing movement before the sense of volition; however, because the timing of the sense of volition has depended on subjective and retrospective judgments, these findings are still regarded with a degree of scepticism. EEG events such as beta event-related desynchronization and movement-related cortical potentials are associated with the brain's programming of movement. Using an optimized EEG signal derived from multiple variables, we were able to make real-time predictions of movements in advance of their occurrence with a low false-positive rate. We asked participants what they were thinking at the time of prediction: Sometimes they were thinking about movement, and other times they were not. Our results indicate that the brain can be preparing to make voluntary movements while participants are thinking about something else.

    View details for DOI 10.1162/jocn_a_00360

    View details for Web of Science ID 000318355000001

    View details for PubMedID 23363409

    View details for PubMedCentralID PMC4747632

  • Intermittent theta-burst transcranial magnetic stimulation for treatment of Parkinson disease NEUROLOGY Benninger, D. H., BERMAN, B. D., Houdayer, E., Pal, N., Luckenbaugh, D. A., Schneider, L., Miranda, S., Hallett, M. 2011; 76 (7): 601-609


    To investigate the safety and efficacy of intermittent theta-burst stimulation (iTBS) in the treatment of motor symptoms in Parkinson disease (PD).Progression of PD is characterized by the emergence of motor deficits, which eventually respond less to dopaminergic therapy and pose a therapeutic challenge. Repetitive transcranial magnetic stimulation (rTMS) has shown promising results in improving gait, a major cause of disability, and may provide a therapeutic alternative. iTBS is a novel type of rTMS that may be more efficacious than conventional rTMS.In this randomized, double-blind, sham-controlled study, we investigated safety and efficacy of iTBS of the motor and dorsolateral prefrontal cortices in 8 sessions over 2 weeks (evidence Class I). Assessment of safety and clinical efficacy over a 1-month period included timed tests of gait and bradykinesia, Unified Parkinson's Disease Rating Scale (UPDRS), and additional clinical, neuropsychological, and neurophysiologic measures.We investigated 26 patients with mild to moderate PD: 13 received iTBS and 13 sham stimulation. We found beneficial effects of iTBS on mood, but no improvement of gait, bradykinesia, UPDRS, and other measures. EEG/EMG monitoring recorded no pathologic increase of cortical excitability or epileptic activity. Few reported discomfort or pain and one experienced tinnitus during real stimulation.iTBS of the motor and prefrontal cortices appears safe and improves mood, but failed to improve motor performance and functional status in PD.This study provides Class I evidence that iTBS was not effective for gait, upper extremity bradykinesia, or other motor symptoms in PD.

    View details for Web of Science ID 000287363800006

    View details for PubMedID 21321333

    View details for PubMedCentralID PMC3053339

  • Prediction of human voluntary movement before it occurs CLINICAL NEUROPHYSIOLOGY Bai, O., Rathi, V., Lin, P., Huang, D., Battapady, H., Fei, D., Schneider, L., Houdayer, E., Chen, X., Hallett, M. 2011; 122 (2): 364-372


    Human voluntary movement is associated with two changes in electroencephalography (EEG) that can be observed as early as 1.5 s prior to movement: slow DC potentials and frequency power shifts in the alpha and beta bands. Our goal was to determine whether and when we can reliably predict human natural movement BEFORE it occurs from EEG signals ONLINE IN REAL-TIME.We developed a computational algorithm to support online prediction. Seven healthy volunteers participated in this study and performed wrist extensions at their own pace.The average online prediction time was 0.62±0.25 s before actual movement monitored by EMG signals. There were also predictions that occurred without subsequent actual movements, where subjects often reported that they were thinking about making a movement.Human voluntary movement can be predicted before movement occurs.The successful prediction of human movement intention will provide further insight into how the brain prepares for movement, as well as the potential for direct cortical control of a device which may be faster than normal physical control.

    View details for DOI 10.1016/j.clinph.2010.07.010

    View details for Web of Science ID 000285995400019

    View details for PubMedID 20675187

    View details for PubMedCentralID PMC5558611