Dr. Esfahanian is a clinical assistant professor of pediatric anesthesiology at Lucile Packard Children's Hospital Stanford. He is board certified in pediatrics, anesthesiology, and pediatric anesthesiology and practices as a pediatric regional anesthesiologist. He has an interest in utilizing regional techniques to enhance postoperative recovery and has presented nationally on the effectiveness of head and neck blocks, particularly for cleft palate repair.

Clinical Focus

  • Anesthesia
  • pediatric anesthesiology
  • pediatric regional anesthesiology

Honors & Awards

  • Cynthia T. Anderson Award: Outstanding performance in the acquisition of medical knowledge., UC Irvine, Dept. of Anesthesiology and Perioperative Care (2018)

Boards, Advisory Committees, Professional Organizations

  • Member, Stanford Pediatric Anesthesia Fellowship Program Education Committee (2018 - Present)
  • Member, Stanford Pediatric Anesthesia Fellowship Program Evaluation Committee (2018 - Present)
  • Diplomate, American Board of Anesthesiology (2019 - Present)
  • Member, American Society of Regional Anesthesia and Pain Medicine (ASRA) (2019 - Present)
  • Member, Society for Pediatric Anesthesia (2018 - Present)
  • Member, American Society of Anesthesiologists (2014 - Present)
  • Member, American Academy of Pediatrics (2013 - Present)

Professional Education

  • Board Certification: American Board of Pediatrics, Pediatrics (2020)
  • Board Certification: American Board of Anesthesiology, Pediatric Anesthesia (2019)
  • Board Certification: American Board of Anesthesiology, Anesthesia (2019)
  • Clinical Scholar, Stanford University Dept. of Anesthesiology, Division of Pediatric Anesthesiology, Pediatric Regional Anesthesia (2020)
  • Fellowship: Stanford University Anesthesiology Fellowships (2019) CA
  • Fellowship, Stanford, Pediatric Anesthesiology (2019)
  • Residency: UC Irvine Combined Anesthesiology/Pediatric Residency (2018) CA
  • Medical Education: Wayne State University School of Medicine (2013) MI
  • Bachelor of Science, Michigan State University, Physics (2008)
  • Bachelor of Science, Michigan State University, Physiology (2008)

Current Research and Scholarly Interests

My current interests include the suprazygomatic maxillary nerve block and its role in enhanced recovery after cleft palate surgery and the development of a high-fidelity ultrasound phantom model to teach this regional anesthesia technique. I am also investigating the role of erector spinae plane blockade in the post-operative recovery of adolescent idiopathic scoliosis patients undergoing posterior spinal fusion.

2020-21 Courses

All Publications

  • Toward Opioid-Free Fast Track for Pediatric Congenital Cardiac Surgery. Journal of cardiothoracic and vascular anesthesia Esfahanian, M., Caruso, T. J., Lin, C., Kuan, C., Purkey, N. J., Maeda, K., Tsui, B. C. 2019

    View details for DOI 10.1053/j.jvca.2019.02.003

    View details for PubMedID 30852093

  • Moving toward patients being pain- and spasm-free after pediatric scoliosis surgery by using bilateral surgically-placed erector spinae plane catheters. Canadian journal of anaesthesia = Journal canadien d'anesthesie Tsui, B. C., Esfahanian, M. n., Lin, C. n., Policy, J. n., Vorhies, J. n. 2019

    View details for DOI 10.1007/s12630-019-01543-0

    View details for PubMedID 31776896

  • Regional changes in cardiac and stellate ganglion norepinephrine transporter in DOCA-salt hypertension. Autonomic neuroscience : basic & clinical Wehrwein, E. A., Novotny, M., Swain, G. M., Parker, L. M., Esfahanian, M., Spitsbergen, J. M., Habecker, B. A., Kreulen, D. L. 2013; 179 (1-2): 99-107


    Uptake of norepinephrine via the neuronal norepinephrine transporter is reduced in the heart during deoxycorticosterone (DOCA)-salt hypertension. We hypothesized that this was due to reduced norepinephrine transporter mRNA and/or protein expression in the stellate ganglia and heart. After 4 weeks of DOCA-salt treatment there was no change in norepinephrine transporter mRNA in either the right or the left stellate ganglia from hypertensive rats (n=5-7, p>0.05). Norepinephrine transporter immunoreactivity in the left stellate ganglion was significantly increased (n=4, p<0.05) while the right stellate ganglion was unchanged (n=4, p>0.05). Whole heart norepinephrine content was significantly reduced in DOCA rats consistent with reduced uptake function; however, when norepinephrine was assessed by chamber, a significant decrease was noted only in the right atrium and right ventricle (n=6, p<0.05). Cardiac norepinephrine transport binding by chamber revealed that it was only reduced in the left atrium (n=5-7, p>0.05). Therefore, 1) contrary to our hypothesis reduced reuptake in the hypertensive heart is not exclusively due to an overall reduction in norepinephrine transporter mRNA or protein in the stellate ganglion or heart, and 2) norepinephrine transporter regulation occurs regionally in the heart and stellate ganglion in the hypertensive rat heart.

    View details for DOI 10.1016/j.autneu.2013.08.070

    View details for PubMedID 24075956

    View details for PubMedCentralID PMC3883044