Bio
Dr. Levin is a board-certified, fellowship-trained ophthalmologist with the Stanford Health Care Byers Eye Institute. He is also a clinical instructor in the Department of Ophthalmology at Stanford University School of Medicine.
Dr. Levin has focused solely on plastic and reconstructive eye surgery since 1992. He evaluates and cares for conditions such as tear duct problems and eyelid problems, including eyelid growths and cancers. As an established expert, he uses the best techniques to improve eye health and confidence in facial appearance.
Dr. Levin has published research findings in many peer-reviewed journals, including the American Journal of Ophthalmology and Archives of Ophthalmology. He has also shared his expertise at numerous ophthalmology conferences and in books such as Contemporary Issues in Ophthalmology and Ophthalmic Surgical Procedures.
He is a fellow of the American Society of Ophthalmic Plastic and Reconstructive Surgery and the American Academy of Ophthalmology. He also has a long history of teaching about ophthalmic procedures at Stanford University School of Medicine.
Clinical Focus
- Ophthalmic Plastic and Reconstructive Surgery
Academic Appointments
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Clinical Instructor, Ophthalmology
Professional Education
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Board Certification: American Board of Ophthalmology, Ophthalmology (1990)
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Fellowship: Duke University Medical Center Ophthalmology Fellowships (1989) NC
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Fellowship: California Pacific Medical Center Dept of Ophthalmology (1988) CA
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Residency: California Pacific Medical Center Dept of Ophthalmology (1987) CA
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Internship: Olive View-UCLA Medical Center Internal Medicine Residency (1984) CA
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Medical Education: Johns Hopkins University School of Medicine (1983) MD
All Publications
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Histopathology of the eye in Cockayne's syndrome.
Archives of ophthalmology (Chicago, Ill. : 1960)
1983; 101 (7): 1093-7
Abstract
The eyes of a 44-month-old boy with Cockayne's syndrome had retinal pigmentary abnormalities that included variable pigmentation and excessive lipofuscin deposition in the retinal pigment epithelium and unusual pigmented cells in the retina and subretinal space. There was optic nerve atrophy with loss of nerve fibers and myelin sheaths and also atrophy of the retinal nerve fiber and ganglion cell layers consistent with the histologic features of a demyelinating disease. Widespread pigment dispersion was found in the anterior segment. There was no evidence of vascular disorder, ocular calcification, neuronal storage disorder, or dystrophic corneal changes.
View details for DOI 10.1001/archopht.1983.01040020095016
View details for PubMedID 6870631
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A clinicopathologic study of optic neuropathies associated with intracranial mass lesions with quantification of remaining axons.
American journal of ophthalmology
1983; 95 (3): 295-306
Abstract
We examined three patients with intracranial mass lesions that damaged the anterior visual pathway and studied this damage histologically after the patients died. Estimation of the number of surviving axons in the six optic nerves generally showed the greatest atrophy to be in the temporal sector in cross sections immediately behind the globe. More posterior to the globe, atrophy appeared greatest in the center of several nerves. This pattern of damage may result from particular vulnerability of macular axons to damage along the anterior visual pathway. Optic disk pallor was present in two eyes that had less than 40% of the normal number of axons remaining, while one optic disk appeared to be normal with 70% of the axons intact. Afferent pupillary defects were observed in two patients who had two to three times as many remaining axons in the contralateral optic nerve as in the nerve on the side of the defect.
View details for DOI 10.1016/s0002-9394(14)78297-2
View details for PubMedID 6829676