Bio

Dr. Sripriya (Priya) Chari is a CA Licensed Clinical Psychologist and Clinical Associate Professor working across the INSPIRE, PTSD and centerspace Clinics at Stanford. Dr. Chari's clinical interests lie in early intervention and providing evidence-based treatments for trauma and psychosis, as well as culturally-attuned services to people from the South Asian diaspora. She is involved in teaching undergraduates (IntroSem on Destigmatizing Psychosis) as well as graduate students (Clinical Perspectives on Trauma Psychology), as well as supervising postdoctoral fellows and practicum students. In addition, she leads outreach efforts into the local South Asian community with a view to educating people about mental health.

Clinical Focus

  • Psychosis
  • Clinical Psychology

Academic Appointments

  • Clinical Associate Professor, Psychiatry and Behavioral Sciences

Professional Education

  • Internship: UC Davis at VA Medical Center Martinez (2014) CA
  • Residency: Santa Clara Medical Center (2016) CA
  • PhD Training: Palo Alto University Pacific Graduate School of Psychology (2014) CA

Contact

  • Academic
    Department: Psych/General Psychiatry and Psychology (Adult) Position: Clinical Associate Professor
  • Clinical (Primary)  INSPIRE Clinic Dept of Psychiatry and Behavioral Sciences 401 Quarry Rd MC 5723 Stanford, CA 94305 (650) 724-9900 (fax)

Additional Clinical Info

Additional Info

2025-26 Courses

All Publications

  • Opening Pandora's Box: The Importance of Assessing and Treating Trauma in Individuals Experiencing Psychosis Psychiatric Times Chari, S., Lee, G. E., Olson, N. D., Hardy, K. V. 2024; 41 (11)
  • I THINK MY PATIENT MAY BE EXPERIENCING PSYCHOSIS - NOW WHAT? HOW PROVIDERS CAN SUPPORT EARLY IDENTIFICATION AND INTERVENTION. Eisen, K., Chari, S., Salaheldin, K., Zepp, C., Ballon, J., Hardy, K. OXFORD UNIV PRESS INC. 2022: S1

    View details for Web of Science ID 000788118600003

  • Impaired prefrontal functional connectivity associated with working memory task performance and disorganization despite intact activations in schizophrenia. Psychiatry research. Neuroimaging Chari, S., Minzenberg, M. J., Solomon, M., Ragland, J. D., Nguyen, Q., Carter, C. S., Yoon, J. H. 2019; 287: 10–18

    Abstract

    Working memory (WM) deficits are key features of schizophrenia and are associated with significant functional impairment. The precise mechanisms of WM and their relationship between WM deficits with other clinical symptoms of schizophrenia remain unclear. Contemporary models propose that WM requires synchronous activity across brain regions within a distributed network, including lateral prefrontal cortex (PFC) and task-relevant posterior sensory cortical regions. This suggests that WM deficits in patients may be due to PFC functional connectivity (FC) impairments rather than activation impairments per se. We tested this hypothesis by measuring the magnitude of FC between lateral PFC and visual cortex and univariate activations within these regions during visual WM. We found decreased FC in patients compared to healthy subjects in the context of similar levels of univariate activity. Furthermore, this decreased FC was associated with task performance and clinical symptomatology in patients. The magnitude of FC, particularly during the delay period, was positively correlated with WM task accuracy, while FC during cue was inversely correlated with severity of disorganization. Taken together, these results suggest that impairment in lateral PFC FC is a key aspect of information processing impairment in patients with schizophrenia, and may be a sensitive index of altered neurophysiology.

    View details for PubMedID 30933745

https://orcid.org/0009-0005-6081-4967