Bio

Dr. Susy Jeng is Clinical Associate Professor of Neurology at Stanford Children’s Hospital at Stanford University. Dr. Jeng received her A.B. at Harvard College and M.D. at the University of California, San Diego. She completed her pediatrics residency at University of California, San Francisco and is board-certified in pediatrics. After practicing general pediatrics for two years, she returned to UCSF for neurology residency. Upon completion of her residencies, she joined the faculty at Stanford as a general child neurologist with a special interest in medical education. She is the site director for the Stanford medical student neurology clerkship and the pediatric neurology liaison to the Stanford pediatrics residency program.

Clinical Focus

  • Neurology with Special Qualifications in Child Neurology

Academic Appointments

Professional Education

  • Board Certification: American Board of Psychiatry and Neurology, Neurology with Special Qualifications in Child Neurology (2011)
  • Board Certification: American Board of Pediatrics, Pediatrics (2006)
  • Residency, UCSF, Child Neurology (2011)
  • Residency, UCSF, Pediatrics (2006)
  • MD, UCSD, Medical School (2003)
  • AB, Harvard College, Biology (1998)

Contact

  • Academic
    sjeng@stanford.edu
    University - Academic staff Department: Pediatric Neurology Position: Clinical Associate Professor
    • 750 Welch Rd Suite 317
    • Palo Alto,  California  94304 
    (650) 723-7299 (fax)
  • Clinical (Primary)  Child Neurology 730 Welch Rd Ste 206 Palo Alto, CA 94304 (650) 721-6350 (fax)

Additional Clinical Info

Additional Info

  • Mail Code: 5235

Links

All Publications

  • Episodic ataxic gait in a healthy toddler: an exemplary case of recurrent vertigo of childhood. The Journal of pediatrics Silverman, A., Barsh, G. R., Jeng, S. 2022

    View details for DOI 10.1016/j.jpeds.2022.12.015

    View details for PubMedID 36549413

  • Diagnosis of Cerebrospinal Fluid Rhinorrhea: Lack of Specificity of the Glucose Oxidase Test Tape JOURNAL OF PEDIATRICS Jeng, S. 2017; 190: 48

    View details for PubMedID 29144269

  • 50 Years Ago in The Journal of Pediatrics: Migraine. journal of pediatrics Jeng, S. 2017; 181: 85-?

    View details for DOI 10.1016/j.jpeds.2016.08.090

    View details for PubMedID 28129878

  • 50 Years ago in the Journal of Pediatrics: a chromosome anomaly in an infant with a degenerative disease of the central nervous system. journal of pediatrics Jeng, S. 2013; 162 (2): 292-?

    View details for DOI 10.1016/j.jpeds.2012.09.004

    View details for PubMedID 23321045

  • A Chromosome Anomaly in an Infant with a Degenerative Disease of the Central Nervous System JOURNAL OF PEDIATRICS Jeng, S. 2013; 162 (2): 292-292

    View details for DOI 10.1016/j.jpeds.2012.09.004

    View details for Web of Science ID 000313579900015

  • Prevalence of Congenital Hydrocephalus in California, 1991-2000 PEDIATRIC NEUROLOGY Jeng, S., Gupta, N., Wrensch, M., Zhao, S., Wu, Y. W. 2011; 45 (2): 67-71

    Abstract

    In a population-based retrospective cohort of 5,353,022 California births from 1991 to 2000, 3,152 newborns were diagnosed with congenital hydrocephalus during the birth hospitalization. We compared demographic and clinical characteristics of infants with and without congenital hydrocephalus, and examined in-hospital fatality rates. The prevalence of congenital hydrocephalus was 5.9 per 10,000. During the study period, there was a decline in congenital hydrocephalus due to spina bifida (1.4 to 0.9 per 10,000), and an increase in congenital hydrocephalus due to obstructive hydrocephalus (0.5 to 1.0 per 10,000). Independent risk factors for congenital hydrocephalus were birth weight <1,500 g (odds ratio [OR] 51.6, 95% confidence interval [CI] 47.7-55.8) and birth weight 1,500-2,000 g (OR 14.1, 95% CI 12.4-16) compared to birth weight greater than 2,000 g, low socioeconomic status (OR 1.5, 95% CI 1.4-1.6), and male sex (OR 1.2, 95% CI 1.1-1.3). Asians had a decreased risk for congenital hydrocephalus (OR 0.7, 95% CI 0.6-0.8) when compared to whites. Thirteen percent of affected neonates died before hospital discharge.

    View details for DOI 10.1016/j.pediatrneurol.2011.03.009

    View details for Web of Science ID 000293260800001

    View details for PubMedID 21763944