Bio

I am a medical scientist with expertise in basic biomedical research focusing on Microbiology and Immunology. After getting medical license from Bangladesh, I received MD in Medical Microbiology from BSMMU, Bangladesh and MS in Environmental Health Science from the University of Tokyo School of Medicine, Japan.

Honors & Awards

  • Precedential award, Bangabandhu Sheikh Mujib Medical University (BSMMU) (2018)

Professional Education

  • B of Medicine and B of Surgery, University Of Dhaka (2013)
  • Master of Health and Science, University Of Tokyo (2023)
  • Doctor of Medicine, Academics (2018)
  • MS, The University of Tokyo, Japan, Environmental Health Science (2023)
  • MD, Bangabandhu Sheikh Mujib Medical University (BSMMU), Bangladesh, Microbiology (2018)
  • MBBS, The University of Dhaka, Bangladesh, Medicine (2012)

Stanford Advisors

Contact

  • Academic
    salvinaz@stanford.edu
    University - Scholar Department: School of Medicine Position: Postdoctoral Scholar
    University - Staff Department: Medicine - Med/Immunology & Rheumatology Position: Life Science Research Professional 1

Additional Info

All Publications

  • Memory B Cell Activation and Dysregulation in Systemic Lupus Erythematosus Younis, S., Moutusy, S., Jahanbani, S., Wu, X., Harris, M., Pandit, M., van Dam, L., Sharpe, O., Utz, P., Robinson, W. WILEY. 2024: 3653-3655

    View details for Web of Science ID 001331419105219

  • Gut Microbiome-Related Anti-Inflammatory Effects of Aryl Hydrocarbon Receptor Activation on Inflammatory Bowel Disease INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES Moutusy, S., Ohsako, S. 2024; 25 (6)

    Abstract

    Inflammatory bowel disease (IBD) is one of the most prevalent chronic inflammations of the gastrointestinal tract (GIT). The gut microbial population, the cytokine milieu, the aryl hydrocarbon receptor (AHR) expressed by immune and nonimmune cells and the intrinsic pathway of Th-cell differentiation are implicated in the immunopathology of IBD. AHR activation requires a delicate balance between regulatory and effector T-cells; loss of this balance can cause local gut microbial dysbiosis and intestinal inflammation. Thus, the study of the gut microbiome in association with AHR provides critical insights into IBD pathogenesis and interventions. This review will focus on the recent advancements to form conceptional frameworks on the benefits of AHR activation by commensal gut bacteria in IBD.

    View details for DOI 10.3390/ijms25063372

    View details for Web of Science ID 001192885500001

    View details for PubMedID 38542367

    View details for PubMedCentralID PMC10970487

https://orcid.org/0009-0001-7206-2185