School of Medicine
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Fanglin Zhang, MD., PhD.
Clinical Assistant Professor, Neurology & Neurological Sciences
BioDr. Zhang is a board-certified, fellowship-trained neurologist. She serves as a clinical assistant professor in the Department of Neurology at Stanford University School of Medicine.
Dr. Zhang diligently excels in diagnosing and treating complex neurological conditions. Her clinical interests span a broad spectrum, with a current emphasis on intractable epilepsy and urgent stroke care. With each patient, Dr. Zhang forms a strong alliance focused on overcoming neurological disease and improving quality of life. She practices a patient-centered and evidence-based medicine.
As a clinician educator, she finds joy in educating both patients and trainees. She also enjoys conducting cutting-edge clinical research. Her work has been published in numerous peer-reviewed journals, such as Neurological Sciences, Annals of Clinical and Translational Neurology, Neuroimage, Glia, Immunology, the Journal of Psychiatric Practice, Journal of Neuroimmunology, etc. Her current research interest includes clinical trials and a large cohort study of the impact of seizures on stroke outcomes.
Among her many honors, Dr. Zhang has won a grant award from the National Multiple Sclerosis Society. Dr. Zhang shows a great enthusiasm in public welfare. She has earned recognition for volunteering her time and expertise at family health centers, hospitals, and schools. -
Hao Zhang
Instructor, Cardiovascular Institute
BioI earned my M.D. from Peking Union Medical College, Tsinghua University. Subsequently, I completed my residency and served as a cardiac fellow at Fuwai Hospital, where I developed a strong foundation in the pathophysiology of cardiovascular diseases.
As a postdoctoral researcher and instructor in Dr. Joseph Wu’s lab at Stanford, I established a protocol to derive cardiac fibroblasts (CFs) from human iPSCs, providing an unlimited source of cells for studying cardiac fibrosis. This method generates homogenous iPSC-derived CFs that remain quiescent yet responsive to profibrotic stimuli. Notably, this protocol played a crucial role in developing a multiscale drug discovery platform that integrates human iPSCs, 3D-engineered heart tissues, and animal models of heart failure. Using this platform, I discovered novel signaling pathways and therapeutic targets for cardiac fibrosis. My most recent work has been published in Cell (https://doi.org/10.1016/j.cell.2024.09.034).
